2748008: Spinal cord structure (body structure)

SNOMED CT Concept\Body structure (body structure)\Anatomical or acquired body structure (body structure)\Anatomical structure\...

\An anatomical structure that consists of the maximal set of organ parts so connected to one another that together they constitute a self-contained unit of macroscopic anatomy, distinct both morphologically and functionally from other such units. Together with other organs, an organ constitutes an organ system or a body part. An organ is divisible into organ parts but not organs (examples: femur, biceps, liver, heart, aorta, sciatic nerve, ovary).\Brain and spinal cord structure (body structure)\Spinal cord structure

\Body region structure\Structure of back of trunk and/or neck (body structure)\This volume includes the spinal column, its spaces and contents, notably the spinal canal, spinal meninges and spinal cord. It also includes the muscles associated with the spine within (and including) the anterior and posterior thoracolumbar fascia and its equivalent nuchal fascia in the neck. This region also includes the spinal ventral (anterior) and dorsal (posterior) roots, the dorsal root (spinal) ganglions and the spinal nerve trunks.\Structure of vertebral column and/or spinal cord (body structure)\Spinal cord structure

Status: current, Not sufficiently defined by necessary conditions definition status (core metadata concept). Date: 31-Jan 2002. Module: SNOMED CT core

Descriptions:

Expanded Value Set

Outbound Relationships	Type	Target	Active	Characteristic	Refinability	Group	Values
Spinal cord structure	Is a	Nervous structure of trunk	false	Inferred relationship	Some
Spinal cord structure	Is a	Spinal structure	false	Inferred relationship	Some
Spinal cord structure	Is a	Spinal cord, roots and ganglia structure	false	Inferred relationship	Some
Spinal cord structure	Is a	Central nervous system part	false	Inferred relationship	Some
Spinal cord structure	partie de	Entire trunk	false	Inferred relationship	Some
Spinal cord structure	partie de	Entire spinal cord, roots and ganglia	false	Inferred relationship	Some
Spinal cord structure	partie de	Entire central nervous system	false	Additional relationship	Some
Spinal cord structure	partie de	Spinal cord, roots and ganglia structure	false	Additional relationship	Some
Spinal cord structure	Is a	Brain and spinal cord structure (body structure)	true	Inferred relationship	Some
Spinal cord structure	partie de	Entire back (surface region)	false	Additional relationship	Some
Spinal cord structure	Is a	Structure of vertebral region of back	false	Inferred relationship	Some
Spinal cord structure	Is a	Structure of vertebral column and/or spinal cord (body structure)	true	Inferred relationship	Some

Inbound Relationships	Type	Active	Source	Characteristic	Refinability	Group
Cyst of spinal cord caused by parasite	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Spinal cord decompression injury	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Disorder due to and following spinal cord injury (disorder)	Finding site	False	Spinal cord structure	Inferred relationship	Some	1
Open fracture of vertebral column with spinal cord injury	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Herpes zoster myelitis (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Closed spinal dislocation with complete lumbar cord lesion	Finding site	False	Spinal cord structure	Inferred relationship	Some	4
Open spinal dislocation with complete thoracic cord lesion	Finding site	False	Spinal cord structure	Inferred relationship	Some	4
Compression of spinal cord due to intraspinal abscess	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Open fracture of sacrum AND/OR coccyx with complete cauda equina lesion	Finding site	True	Spinal cord structure	Inferred relationship	Some	4
Traumatic spinal cord haemorrhage	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
Myelopathy due to vitamin B12 deficiency	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myelopathy due to lathyrism (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Granuloma of spinal cord caused by parasite	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Spinal cord compression due to granulomatous disorder	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Granuloma of spinal cord caused by bacterium (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Granuloma of spinal cord caused by Mycobacterium (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Spinal cord compression due to cavernous haemangioma	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Encephalomyelocele	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
Injuries of brain and cranial nerves with injuries of nerves and spinal cord at neck level	Finding site	True	Spinal cord structure	Inferred relationship	Some	4
Cavernous haemangioma of spinal cord	Finding site	False	Spinal cord structure	Inferred relationship	Some	1
Implantation of neurostimulator device of spinal cord	Procedure site - Indirect (attribute)	True	Spinal cord structure	Inferred relationship	Some	1
Familial spinal neurofibromatosis	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Lipoma due to neurospinal dysraphism	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Spina bifida without hydrocephalus - open	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
Thoracic spina bifida without hydrocephalus - open	Finding site	False	Spinal cord structure	Inferred relationship	Some	3
Sacral spina bifida without hydrocephalus - open	Finding site	False	Spinal cord structure	Inferred relationship	Some	2
Cervical spina bifida without hydrocephalus - open	Finding site	False	Spinal cord structure	Inferred relationship	Some	3
Spina bifida with hydrocephalus - open	Finding site	True	Spinal cord structure	Inferred relationship	Some	5
Rachischisis is a neural tube defect, which occurs when the neural folds do not join at the midline and the undifferentiated neuroectoderm remains exposed. Rachischisis totalis (holorachischisis) is the extreme form in which the entire spinal cord remains open.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Rachischisis is a neural tube defect, which occurs when the neural folds do not join at the midline and the undifferentiated neuroectoderm remains exposed. Rachischisis partialis (merorachischisis) is the partial form where the spinal cord is partially closed and partially open.	Finding site	True	Spinal cord structure	Inferred relationship	Some	2
A rare closed neural tube defect characterized by cystic dilatation of the central canal of the spinal cord, herniating through a posterior vertebral arch defect (spina bifida) into an expanded cerebrospinal fluid (CSF) filled dural sac (meningocele). It can be located at the caudal part of the spinal cord (terminal myelocystocele) or above conus (non-terminal myelocystocele).	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
Hemimyelocele	Finding site	True	Spinal cord structure	Inferred relationship	Some	4
Cervical myelocele	Finding site	False	Spinal cord structure	Inferred relationship	Some	5
Cervical hydromyelocele	Finding site	False	Spinal cord structure	Inferred relationship	Some	6
Thoracic hydromyelocele	Finding site	False	Spinal cord structure	Inferred relationship	Some	6
Lumbar myelocele	Finding site	False	Spinal cord structure	Inferred relationship	Some	5
Thoracic myelocele	Finding site	False	Spinal cord structure	Inferred relationship	Some	5
Spontaneous hematoma of spinal cord (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Traumatic spinal cord injury (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myelopathy due to folate deficiency	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myxopapillary ependymoma (MEPN) describes a slow growing ependymoma located almost exclusively in the conus medullaris-cauda equina-filum terminale region of the spinal cord, presenting in all age groups, and manifesting with variable symptoms such as neck pain, vomiting and unsteady gait and metastasis. It has a more aggressive disease course and is seen in the paediatric population.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
X-linked complex hereditary spastic paraplegia	Finding site	False	Spinal cord structure	Inferred relationship	Some	2
X-linked pure hereditary spastic paraplegia	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Gliosarcoma of spinal cord (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare subtype of split cord malformation characterised by each hemicord contained in its own dural sac, typically with an intervening bony septum.	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
A rare complex hereditary spastic paraplegia characterized by neonatal to infantile onset of progressive spasticity in the lower limbs, hyperreflexia, tip-toe walking, pes equinus, and delayed motor developmental milestones. Kyphoscoliosis becomes evident in older patients, and most patients show atrophy of the lateral aspects of the tongue. Additional signs may include intellectual disability, language impairment, and moderate upper limb involvement.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Autosomal recessive spastic paraplegia type 76 is a rare, complex hereditary spastic paraplegia characterized by adult onset slowly progressive, mild to moderate lower limb spasticity and hyperreflexia, resulting in gait disturbances, commonly associated with upper limb hyperreflexia and dysarthria. Foot deformities (usually pes cavus) and extensor plantar responses are also frequent. Additional features may include ataxia, lower limb weakness/amyotrophy, abnormal bladder function, distal sensory loss and mild intellectual deterioration.	Finding site	True	Spinal cord structure	Inferred relationship	Some	2
Allan-Herndon-Dudley syndrome	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare genetic neurological disorder characterized by postnatal microcephaly, hypotonia during infancy followed in most cases by progressive spasticity mainly affecting the lower limbs, and spastic diplegia or paraplegia, intellectual disability, delayed or absent speech, and dysarthria. Seizures and mildly dysmorphic features have been described in some patients.	Finding site	True	Spinal cord structure	Inferred relationship	Some	4
Spinal cord compression due to tuberculosis (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Spinal cord compression due to parasitic disease (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare autosomal recessive complex spastic paraplegia characterized by mostly adult-onset progressive spasticity and weakness predominantly affecting the lower limbs, axonal motor and sensory neuropathy, and cerebellar symptoms like ataxia, dysarthria, and oculomotor abnormalities. Variable degrees of cognitive impairment may also be present. Subtle extrapyramidal involvement and supranuclear gaze palsy were reported in some cases. Features on brain imaging include cerebral and cerebellar atrophy and sometimes abnormalities of the corpus callosum or basal ganglia.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Encephalomyelitis caused by Schistosoma (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Mycobacterial spinal cord abscess	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Fungal spinal cord compression	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare genetic neurological disorder characterized by progressive spastic paraparesis and delayed gross motor development with an onset in infancy or early childhood. Patients also show variable degrees of intellectual disability, speech delay, and dysarthria. Other reported features include microcephaly, seizures, bifid uvula with or without cleft palate, and ocular anomalies. Brain imaging shows white matter abnormalities in the periventricular and other regions.	Finding site	False	Spinal cord structure	Inferred relationship	Some	2
Myelopathy due to copper deficiency	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Disorder of spinal cord due to and following intrathecal injection of chemotherapeutic agent (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	2
Myelopathy caused by spider venom	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myelopathy due to and following venomous injury (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myelopathy due to excess zinc	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Disorder of spinal cord due to excess nutrient (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myelopathy due to fluorosis	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Myelopathy due to food toxicity	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Spinocerebellar ataxia due to vitamin E deficiency (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	2
Snake venom-induced myelopathy	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Implantation of electronic stimulator of spine	Procedure site	True	Spinal cord structure	Inferred relationship	Some	1
Disorder of spinal cord caused by cassava (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Disorder of spinal cord due to obesity (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	2
Autosomal recessive spastic paraplegia type 74 is a rare, genetic, spastic paraplegia-optic atrophy-neuropathy-related (SPOAN-like) disorder characterized by childhood onset of mild to moderate spastic paraparesis which manifests with gait impairment that very slowly progresses into late adulthood, hyperactive patellar reflex and bilateral extensor plantar response, in association with optic atrophy and typical symptoms of peripheral neuropathy, including reduced or absent ankle reflexes, lower limb atrophy and distal sensory impairment. Reduced visual acuity and pes cavus are frequently reported.	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
Removal of intrathecal drug delivery device adjacent to spinal cord (procedure)	Procedure site - Indirect (attribute)	True	Spinal cord structure	Inferred relationship	Some	1
Attention to intrathecal drug delivery device adjacent to spinal cord (procedure)	Procedure site - Indirect (attribute)	True	Spinal cord structure	Inferred relationship	Some	1
Spastic paraplegia-severe developmental delay-epilepsy syndrome is a rare, genetic, complex spastic paraplegia disorder characterized by an infantile-onset of psychomotor developmental delay with severe intellectual disability and poor speech acquisition, associated with seizures (mostly myoclonic), muscular hypotonia which may be noted at birth, and slowly progressive spasticity in the lower limbs leading to severe gait disturbances. Ocular abnormalities and incontinence are commonly associated. Other symptoms may include verbal dyspraxia, hypogenitalism, macrocephaly and sensorineural hearing loss, as well as dystonic movements and ataxia with upper limb involvement.	Finding site	True	Spinal cord structure	Inferred relationship	Some	4
Autosomal dominant hereditary spastic paraplegia	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
X-linked hereditary spastic paraplegia (disorder)	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Autosomal recessive hereditary spastic paraplegia	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Pure hereditary spastic paraplegia	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
A rare, pure or complex form of hereditary spastic paraplegia characterized by either a pure spastic paraplegia phenotype, usually presenting in the first or second decade of life, with spastic lower extremities, unsteady spastic gait, hyperreflexia and extensor plantar responses, or as a complicated phenotype with the additional manifestations of distal wasting, saccadic ocular movements, mild cerebellar ataxia and mild, distal, axonal neuropathy.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Autosomal recessive spastic paraplegia type 35 is a rare form of hereditary spastic paraplegia characterized by childhood (exceptionally adolescent) onset of a complex phenotype presenting with lower limb (followed by upper limb) spasticity with hyperreflexia and extensor plantar responses, with additional manifestations including progressive dysarthria, dystonia, mild cognitive decline, extrapyramidal features, optic atrophy and seizures. White matter abnormalities and brain iron accumulation have also been observed on brain magnetic resonance imaging.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A pure or complex form of hereditary spastic paraplegia characterized by an onset in the first decade of life of spastic paraparesis (more prominent in lower than upper extremities) and unsteady gait, as well as increased deep tendon reflexes, amyotrophy, cerebellar ataxia, and flexion contractures of the knees, in some.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Hereditary spastic paraplegia	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
A rare genetic neurological disorder characterized by infantile to childhood onset of progressive sensory neuropathy in association with spastic paraplegia and mutilating acropathy. Patients present lower limb spasticity and progressive severe sensory loss leading to chronic ulcerations in both upper and lower limbs. Electrophysiological studies are consistent with axonal sensory neuropathy, and nerve biopsy shows axonopathy with loss of myelinated nerve fibers of all diameters as well as of unmyelinated axons.	Finding site	True	Spinal cord structure	Inferred relationship	Some	2
Spastic paraplegia-precocious puberty syndrome is a complex form of hereditary spastic paraplegia characterized by the onset of progressive spastic paraplegia associated with precocious puberty (due to Leydig cell hyperplasia) in childhood (at the age of 2 years). Moderate intellectual disability was also reported. There have been no further descriptions in the literature since 1983.	Finding site	True	Spinal cord structure	Inferred relationship	Some	4
A rare genetic neurological disorder characterized by progressive spastic paraparesis and delayed gross motor development with an onset in infancy or early childhood. Patients also show variable degrees of intellectual disability, speech delay, and dysarthria. Other reported features include microcephaly, seizures, bifid uvula with or without cleft palate, and ocular anomalies. Brain imaging shows white matter abnormalities in the periventricular and other regions.	Finding site	True	Spinal cord structure	Inferred relationship	Some	3
X-linked complex hereditary spastic paraplegia	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare complex hereditary spastic paraplegia characterized by adulthood-onset of slowly progressive, bilateral, mainly lower limb spasticity and distal weakness associated with lower limb pain, hyperreflexia, and reduced vibration sense. Axonal neuropathy is frequently observed on electromyography and nerve conduction examination.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A pure form of hereditary spastic paraplegia characterized by a childhood- to adulthood-onset of slowly progressive lower limb spasticity and hyperreflexia of lower extremities, extensor plantar reflexes, distal sensory impairment, variable urinary dysfunction and pes cavus.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A pure form of hereditary spastic paraplegia characterized by a slowly progressive and relatively benign spastic paraplegia presenting in adulthood with spastic gait, lower limb hyperreflexia, extensor plantar responses, bladder dysfunction (urinary urgency and/or incontinence), and mild sensory and motor peripheral neuropathy.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Autosomal recessive spastic paraplegia type 28 is a pure form of hereditary spastic paraplegia characterized by a childhood or adolescent onset of slowly progressive, pure crural muscle spastic paraparesis which manifests with mild lower limb weakness, gait difficulties, extensor plantar responses, and hyperreflexia of lower extremities. Less common manifestations include cerebellar oculomotor disturbance with saccadic eye pursuit, pes cavus and scoliosis. Some patients also present pin and vibration sensory loss in distal legs.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A complex form of hereditary spastic paraplegia characterized by delays in motor development followed by a slowly progressive spastic paraplegia (affecting mainly lower extremities) associated with a desquamating facial rash with butterfly distribution (presenting at around two months of age) and dysarthria. There have been no further descriptions in the literature since 1982.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare type of hereditary spastic paraplegia usually characterized by a pure phenotype of proximal weakness of the lower extremities with spastic gait and brisk reflexes, with a bimodal age of onset of either childhood or adulthood (>30 years). In some cases, it can present as a complex phenotype with additional associated manifestations including peripheral neuropathy, bulbar palsy (with dysarthria and dysphagia), distal amyotrophy, and impaired distal vibration sense.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A pure form of hereditary spastic paraplegia characterized by slowly progressive spastic paraplegia of lower extremities with an age of onset ranging from childhood to adulthood and patients presenting with spastic gait, increased tendon reflexes in lower limbs, extensor plantar response, weakness and atrophy of lower limb muscles and, in rare cases, pes cavus. No abnormalities are noted on magnetic resonance imaging.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A pure form of hereditary spastic paraplegia characterized by a childhood- to adulthood-onset of slowly progressive spastic gait, extensor plantar responses, brisk tendon reflexes in arms and legs, decreased vibration sense at ankles and urinary dysfunction. Ankle clonus is also reported in some patients.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Intellectual disability-spasticity-ectrodactyly syndrome is a rare intellectual disability syndrome characterized by severe intellectual disability, spastic paraplegia (with wasting of the lower limbs) and distal transverse defects of the limbs (e.g. ectrodactyly, syndactyly, clinodactyly of the hands and/or feet).	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare complex hereditary spastic paraplegia characterized by juvenile to adult onset of slowly progressive spasticity mainly affecting the lower limbs, associated with spastic dysarthria and motor neuropathy. Additional manifestations include congenital bilateral cataract, gastroesophageal reflux, persistent vomiting, mild cerebellar signs, pes cavus, and occasionally short stature, among others.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare predominantly pure hereditary spastic paraplegia characterized by juvenile or adult onset of slowly progressive spastic paraparesis, gait disturbances, and increased tendon reflexes. Additional variable manifestations include pes cavus, dysarthria, sensory impairment, and urinary symptoms. Cognition is normal.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare complex hereditary spastic paraplegia characterized by early onset of slowly progressive spastic para- or tetraparesis, increased tendon reflexes, positive Babinski sign, global developmental delay, cognitive impairment, and pseudobulbar palsy. Additional manifestations include dysmorphic facial features, tremor, short stature, and urinary incontinence.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A pure form of hereditary spastic paraplegia characterized by adult onset of crural spastic paraparesis, hyperreflexia, extensor plantar responses, proximal muscle weakness, mild muscle atrophy, decreased vibration sensation at ankles, and mild urinary dysfunction. Foot deformities have been reported to eventually occur in some patients. No abnormalities are noted on brain magnetic resonance imaging and peripheral nerve conduction velocity studies.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare, complex hereditary spastic paraplegia characterized by an early onset and slow progression of spastic paraplegia associated with cerebellar signs, nystagmus, peripheral neuropathy, extensor plantar responses and borderline to mild intellectual disability. Additional features of hypo- or areflexia, mild upper limb involvement and significant visual impairment (optic atrophy, vision loss, astigmatism) have been reported.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
Autosomal recessive spastic paraplegia type 77 is a rare, pure or complex hereditary spastic paraplegia characterized by an infancy to childhood onset of slowly progressive lower limb spasticity, delayed motor milestones, gait disturbances, hyperreflexia and various muscle abnormalities, including weakness, hypotonia, intention tremor and amyotrophy. Ocular abnormalities (e.g. strabismus, ptosis) and other neurological abnormalities, such as dysarthria, seizures and extensor plantar responses, may also be associated.	Finding site	True	Spinal cord structure	Inferred relationship	Some	1
A rare autosomal recessive axonal hereditary motor and sensory neuropathy characterized by infantile onset of recurrent episodes of acute liver failure (resulting in chronic liver fibrosis and hepatosplenomegaly), delayed motor development, cerebellar dysfunction presenting as gait disturbances and intention tremor, neurogenic stuttering, and motor and sensory neuropathy with muscle weakness especially in the lower legs, and numbness. Mild intellectual disability was reported in some patients. MRI of the brain shows non-progressive atrophy of the cerebellar vermis and thinning of the optic nerve.	Finding site	True	Spinal cord structure	Inferred relationship	Some	2

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Reference Sets

Anatomy structure and entire association reference set (foundation metadata concept)

Anatomy structure and part association reference set (foundation metadata concept)

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Id	Description	Lang	Type	Status	Case?	Module
5644011	Spinal cord	en	Synonym (core metadata concept)	Active	Entire term case insensitive (core metadata concept)	SNOMED CT core
483549018	Spinal cord structure	en	Synonym (core metadata concept)	Active	Entire term case insensitive (core metadata concept)	SNOMED CT core
758052016	Spinal cord structure (body structure)	en	Fully specified name	Active	Entire term case insensitive (core metadata concept)	SNOMED CT core
921171000172113	medulla spinalis	fr	Synonym (core metadata concept)	Active	Entire term case insensitive (core metadata concept)	SNOMED CT Switzerland NRC maintained Module
926191000172117	moelle épinière	fr	Synonym (core metadata concept)	Active	Entire term case insensitive (core metadata concept)	SNOMED CT Switzerland NRC maintained Module
966921000172114	moelle spinale	fr	Synonym (core metadata concept)	Active	Entire term case insensitive (core metadata concept)	SNOMED CT Switzerland NRC maintained Module
1017361000172114	structure de la moelle spinale	fr	Synonym (core metadata concept)	Active	Entire term case insensitive (core metadata concept)	SNOMED CT Switzerland NRC maintained Module