48707006: Growth acceleration (morphologic abnormality)

SNOMED CT Concept\Body structure (body structure)\Morphologically altered structure\Morphologically abnormal structure\Growth alteration\Growth acceleration

Status: current, Not sufficiently defined by necessary conditions definition status (core metadata concept). Date: 31-Jan 2002. Module: SNOMED CT core

Descriptions:

Id Description Lang Type Status Case? Module

81149017 Growth acceleration en Synonym (core metadata concept) Active Entire term case insensitive (core metadata concept) SNOMED CT core

81152013 Increased growth en Synonym (core metadata concept) Active Entire term case insensitive (core metadata concept) SNOMED CT core

786305015 Growth acceleration (morphologic abnormality) en Fully specified name Active Entire term case insensitive (core metadata concept) SNOMED CT core

Expanded Value Set

Outbound Relationships	Type	Target	Active	Characteristic	Refinability	Group	Values
Growth acceleration	Is a	Growth alteration	true	Inferred relationship	Some

Inbound Relationships	Type	Active	Source	Characteristic	Refinability	Group
Hypersomatotropic gigantism	Associated morphology	False	Growth acceleration	Inferred relationship	Some	1
Hypersomatotropic gigantism	Associated morphology	False	Growth acceleration	Inferred relationship	Some	1
X-linked acrogigantism	Associated morphology	False	Growth acceleration	Inferred relationship	Some	1
A rare, genetic pituitary disease characterized by infantile-onset, rapid and excessive acceleration of linear growth and body size due to mixed growth hormone (GH)- and prolactin-secreting adenomas and/or pituitary hyperplasia. Patients present with gigantism and may have associated acromegalic features (e.g. coarse facial features, frontal bossing, prognathism, increased interdental space) as well as marked enlargement of hands and feet, soft tissue swelling, increased appetite and acanthosis nigricans.	Associated morphology	True	Growth acceleration	Inferred relationship	Some	1
A type of familial infantile gigantism caused by microduplication of Xq26.3. Onset usually occurs in the first year of life in previously normal infants. Patients present with gigantism and may associate acromegalic features (e.g. coarse facial features, frontal bossing, prognathism, increased interdental space) as well as marked enlargement of hands and feet, soft tissue swelling, appetite increase and acanthosis nigricans. May present as a sporadic condition or as familial isolated pituitary adenomas.	Associated morphology	True	Growth acceleration	Inferred relationship	Some	4
Large for gestation age fetus	Associated morphology	True	Growth acceleration	Inferred relationship	Some	1

Reference Sets